Overprinting translational domains in passive margin salt basins: insights from analogue modelling

Ge, Zhiyuan; Rosenau, Matthias; Warsitzka, Michael; Gawthorpe, Rob L.

doi:https://doi.org/10.5194/se-10-1283-2019

Articles | Volume 10, issue 4

https://doi.org/10.5194/se-10-1283-2019

© Author(s) 2019. This work is distributed under
the Creative Commons Attribution 4.0 License.

https://doi.org/10.5194/se-10-1283-2019

© Author(s) 2019. This work is distributed under
the Creative Commons Attribution 4.0 License.

Articles | Volume 10, issue 4

Research article

|

02 Aug 2019

Research article |

| 02 Aug 2019

Overprinting translational domains in passive margin salt basins: insights from analogue modelling

Zhiyuan Ge, Matthias Rosenau, Michael Warsitzka, and Rob L. Gawthorpe

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Final revised paper (published on 02 Aug 2019)
Preprint (discussion started on 14 Jan 2019)

Interactive discussion

Status: closed

AC: Author comment | RC: Referee comment | SC: Short comment | EC: Editor comment

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- Supplement

RC1: 'Review manuscript se-201902', Frank Zwaan, 27 Mar 2019
- AC1: 'Reply to reviewer 1', Zhiyuan Ge, 27 May 2019
RC2: 'Ge et al. ms', Tim Dooley, 02 Apr 2019
- AC2: 'Reply to reviewer 2', Zhiyuan Ge, 27 May 2019

Peer-review completion

AR: Author's response | RR: Referee report | ED: Editor decision

AR by Zhiyuan Ge on behalf of the Authors (18 Jun 2019) Author's response Manuscript

ED: Publish as is (25 Jun 2019) by Mark Allen

ED: Publish as is (25 Jun 2019) by Federico Rossetti (Executive editor)

AR by Zhiyuan Ge on behalf of the Authors (26 Jun 2019) Manuscript

Short summary

Salt basins are important as they bear abundant natural resources and record valuable geological information. However, some models of salt basin evolution do not fully reconcile with natural examples. Using state-of-the-art analogue modelling, we investigate how a relatively stable area, the translational domain, occurs and gets overprinted in salt basins. The results suggest that that variation of sediment deposition is the key factor for overprinting the translational domain.